General Discussion
Related: Editorials & Other Articles, Issue Forums, Alliance Forums, Region ForumsWanna end the mask war?
SCIENTISTS tell us this virus attacks blood vessels, veins and capillaries leading to long term damage to especially lungs, organ with most vessels, the heart, kidneys and other organs. Name another Organ that is totally dependent on its function of blood vessels. The penis. So Bloomberg et al should start a media blitz of dead and dying penises of Covid victims. Even a pill can not pump blood into an organ with damaged, Shriveled, vessels. I have also seen reports of amputation of limbs from lack of blood flow from severe Covid damage. ALL men would begin wearing double N95 immediately, and the Kens will demand their Karens wear masks to protect their junk. Just saying!
Rorey
(8,445 posts)I went to Walmart a few days ago and they had removed all of the directional signage to have people go just one way in the aisles, and hand sanitizer was on clearance for a dollar.
So I guess it's over.
Maybe they will begin selling Chinese caskets to their shopperss lost family. Or Urns, with Trump 2020 on them for ashes. Walmart could lose a large portion of its customers when this second wave blows up 50 percent higher than it already is in Europe. Herd Immunity....Herd mentality.....neither, it is herd thinning. Gonna be a large loss of tax revenue to pay roll Congress, White House, and subsidize billionaires. MAGA.
Rorey
(8,445 posts)Not sure where they're made.
Traildogbob
(8,834 posts)Probably not made in MurKKKa.
Tanuki
(14,924 posts).... "Introduction
The global outbreak of coronavirus disease (COVID-19) caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) represents an unprecedented challenge for healthcare. Despite social distancing and isolation measures, the number of affected patients is growing daily. Hyperinflammation and immunosuppression are prominently featured in COVID-19 [1, 2], resulting in a cytokine storm [3] ultimately leading to development of micro-thrombosis and disseminated intravascular coagulation (DIC). This cytokine storm is strongly associated with the development of interstitial pneumonia (IP) [4]; however, although lungs are the primarily targeted organs, the cardiovascular system is globally affected. Evidence in this regard supports the notion that the exaggerated production of early response proinflammatory cytokines, such as tumor necrosis factor (TNF), interleukin-1β, -6, and -10 (IL-1β, IL-6, and IL-10, respectively), increases the risk of vascular hyperpermeability, possibly progressing to multiple organ failure and, ultimately, death [3]. The presence of vascular dysfunction at multiple levels, including pulmonary embolisms, alveolar hemorrhage, microangiopathy and vasculitis has been ascertained in post-mortem examination [5, 6]. Additionally, both venous and arterial thromboembolic complications, including endothelial inflammation, have been reported [7, 8]. Indeed, a growing body of evidence seems to support the theory that the endothelium is targeted by the SARS-CoV-2 [9]; most importantly, the endothelium expresses the protein angiotensin-converting enzyme 2 (ACE2) [10, 11], through which the virus can access host cells [12]. Endothelial dysfunction is, therefore, a pivotal determinant of COVID-19 symptoms [13, 14].
As of June 12th, 2020, more than 7.5 million COVID-19 cases have been confirmed worldwide, with more than 420,000 lives lost due to the disease [15]. More than 3.5 million subjects have recovered from COVID-19; however, the long-term consequences of the disease are still largely unknown. Data from 20022004 epidemics of SARS suggest that cardiovascular sequelae, such as microangiopathy, cardiomyopathy and impaired endothelial function, are to be expected also in COVID-19 patients [5, 16]. However, while similarities with SARS have been identified, COVID-19 is largely more prevalent due to its high transmissibility, and its consequences, even for recovered patients, are likewise more worrying. Additionally, new evidence is suggesting that autoimmune conditions, such as type 1 diabetes mellitus, might be triggered by the onset of COVID-19 [17], therefore, worsening the risk profile for survivors.
These findings can be extremely relevant for male sexual health: indeed, based on these premises, there is quite enough evidence to hypothesize that consequences of COVID-19 can extend to sexual and reproductive health. We investigated the current literature to understand the long-term clinical complications for COVID-19 survivors, aiming to provide adequate information for clinicians to plan adequate and timely intervention measures."...(more)
Traildogbob
(8,834 posts)Much more eloquent than my theory post. Incredible explanation. But for the trump bubbas, we need to make it easy to understand. You could lose your dick. Then, that would mean a mass increase of big gun purchases. Seriously thank you for that data.
Science Matters
Tanuki
(14,924 posts)cut to the chase! They don't care about science and big words, but the thought of seeing their penis shrivel up and fall off will get their attention!
🍄 🤯
Aristus
(66,478 posts)But it was a good idea...
Doreen
(11,686 posts)Up they will not be reproducing more of themselves.
cayugafalls
(5,646 posts)Seems to be working as they don't say anything back except, "Oh yea..."
GoCubsGo
(32,097 posts)Was she wearing her husband's junk as some sort of trophy?