Last night something came up that made me pull out Mike Vreeland's letter from pre-911. Assuming the entire thing wasn't a hoax and I think enough of us were around AT THE TIME so watched the whole thing develop and analyzed it...
I did 2 searches. One, unrelated to this thread was for the M234 RAG with kinetic projectile launcher www.dtic.mil/ndia/nld4/flat.pdf &
https://192.156.75.103/mcrp/view/mcrp337c/mcrp337c.pdf and the second quick search was for "brown recluse" because that has ALWAYS nagged at me about Vreeland's message. So I read a little and then clicked on this thread because the board is very slow this morning. Shit. Did a google on "brown recluse" meningitis
http://www.google.com/search?sourceid=navclient&ie=UTF-8&oe=UTF-8&q=%22meningitis%22+%22brown+recluse%22and found this:
was apparently bitten on June 2. I remember feeling pain at the site, but didn't really pay much attention at the time, as I was busy rescuing a kitten that was stuck under the garage.
I developed a severe headache and stiff neck that evening that progressed over the next day, sending me to the ER thinking I might have meningitis. There, blood cultures were drawn and found to be negative. I was put on Augmentin (for precaution, I guess). There was no improvement in my general sympotms. The ankle site remained red, then blistered, then red with black center that was surrounded by a red ring. I still didn't think about a BRB, thinking instead it was an infected chigger bite.
To make a really long story shorter, after no less than 4 doctor visits (after the inital ER visit), my neighbor discovered the cause. I had experienced 2 full days of nausea and vomiting, along with severe joint pain and muscle cramping, headache and stiff neck. They came over to check on me and she happened to see the ulcer on my ankle, which she said looked like a Fiddleback bite. She got her husband (a paramedic) to look at it....and off we went to the ER again. After IV Zithromax, Phenergan, and 1000cc of IV fluid, I'm beginning to feel somewhat human again. The ER physician diagnosed it as a brown spider bite and has had me keep my leg elevated with hot moist packs, antibiotic ointment and has continued me on Zithromax by mouth.
The ulcer hasn't increased in size (which is approx. 1cm in diameter)and the black center is no longer there....it is now pink but still cratered. It is also less sore than it was. http://www.highway60.com/mark/brs/bite.asp?Msg=489-----
Cutaneous Anthrax Management Algorithm
1. Suspicious Lesions
a. The highest suspicion should be given to those lesions where the patient had a known or highly suspected exposure to anthrax. However, as some of the recent cases have demonstrated, no known exposure had occurred when the patients presented for care.
b. After an incubation period of approximately 7 days (range = 1 to 12 days), cutaneous anthrax begins as a papule, usually on an exposed area, such as the head, neck, or an upper extremity. The papule may resemble an insect or spider bite and may itch.
c. The papule enlarges and develops a central vesicle or bulla with surrounding brawny, non-pitting edema.
d. The central vesicle becomes hemorrhagic, depressed, and necrotic, and it may become surrounded by satellite vesicles.
e. A central black eschar forms, and the surrounding erythema and edema increase. The necrotic ulcer is usually painless, which is an important differentiating feature from a brown recluse spider bite.
Pustules are rarely present in anthrax lesions, and a primary pustular lesion is unlikely to be cutaneous anthrax. Primary lesions presenting as cloudy vesicles may occur.
f. Lesions progress from papule to vesicle to ulcer to eschar with or without antibiotic therapy as the progression is based on toxin production.
g. Lesions may be solitary or multiple, and if multiple, they are usually found on the same part of the body.
h. Tender regional lymphadenopathy, fatigue, fever, and/or chills may accompany cutaneous findings (ulceroglandular disease).http://216.239.57.104/search?q=cache:xVtfObA5KTQJ:www.aad.org/BioInfo/Biomessage2.html+%22meningitis%22+%22brown+recluse%22&hl=en&ie=UTF-8----
Bioterrorism Summaries from Annual Session 2002Course Title: Bioterrorism
Section: Disaster Preparedness
Faculty Member: Richard P. Wenzel, MSc, MACP
Date/time: April 11, 2002, 10:45 a.m.-12:15 p.m.
Course Number: MTP 131
Reporter: Shannon Donovan, MA
Introduction
Timely recognition of symptoms and early treatment are key to the survival of victims of bioterror attacks. Physicians must be able to diagnose quickly the symptoms of such attacks.
<snip>
Clinical Questions
What are the clinical characteristics of infection with various biological agents that may be used as weapons?
What is the pathogenesis of anthrax, smallpox, and botulism?
What measures can be taken to control the problem?
Key Points
Symptoms of anthrax are nonspecific: fever, chills, fatigue, and malaise.
Anthrax is characterized by hemorrhage and edema. Key symptoms of cutaneous anthrax are massive edema and painless ulcers (unlike those seen after brown recluse spider bites).
Only 8 anthrax spores have to reach the mediastinal nodes to cause pleural effusions, followed by bloodstream infection, sepsis and septic shock, and (in 50% of patients) meningitis.
Hemorrhagic meningitis occurs in 50% of patients and should be treated expectantly with antibiotics that cross the blood-brain barrier.
Gross pathologic examination of patients with anthrax reveals hemorrhage, edema, hemorrhagic necrosis of the thoracic lymph nodes, hemorrhagic mediastinitis, focal hemorrhage at portal of entry, and hematogenous spread (meningitis <"cardinal's cap"> and gastrointestinal tract infection)
----
and this:
Bioterrorism: Background and High Priority Agents
<snip>
Inhalational anthrax is the most lethal form of anthrax. The incubation period typically ranges from 1 to 7 days but may last up to 60 days. Initial symptoms include mild fever, muscle aches, and malaise. Inhalational anthrax has a high case-fatality rate, progressing to respiratory failure (with radiographic evidence of mediastinal widening) and shock. Cutaneous anthrax is the most common form of the disease and appears to require lower doses of spores rubbed into the skin or introduced into cuts in the skin. The incubation period ranges from 1 to 12 days.
A skin lesion evolving from a papule, through a vesicular stage, to a depressed black eschar, characterizes cutaneous anthrax. The lesion is usually painless, but patients may also have fever, malaise, headache, and regional lymphadenopathy. (Note: Recent cutaneous anthrax cases had lesions clinically identified as “Brown Recluse bites”). Gastrointestinal anthrax usually follows after eating raw or undercooked contaminated meat and can have an incubation period of 1 to 7 days. Symptoms are severe abdominal pain followed by fever and signs of septicemia. Lower bowel inflammation typically causes nausea, loss of appetite, and fever followed by abdominal pain, hematemesis, and bloody diarrhea.
http://www.acponline.org/bioterro/as_sum2.htmIs it time to re-examine Vreeland? Seeing just how much migh or might not add up?
http://www.gaianxaos.com/SpecialReports_files/vreelandnote.htm