Fanning the unseen flames of illness

[HysteryDiagnosis]

a potential culprit in the development of AZ, heart disease, (no doubt) diabetes, and most likely cancer... oh and weight gain, I should mention that as well, (Syndrome X)

More at link: http://newsguide.us/index.php?path=/health-medical/cancer/Is-Inflammation-Causing-Cancer-Arthritis-and-Alzheimer-s/

We tend to think of inflammation as synonymous with arthritis. However, inflammation can strike anywhere in your body. Harvard-trained Vijay Nair, MD has researched inflammation and found that it can cause diseases no one wants to get, like cancer, arthritis, heart disease, digestive tract diseases, macular degeneration, Alzheimer's disease, and chronic fatigue syndrome.

Sheffield, MA December 29, 2008 -- Inflammation is your immune system's way of protecting you from physical trauma or foreign invaders, such as viruses and bacteria. It's supposed to be a short and powerful response. Unfortunately, the body doesn't turn off the inflammation switch and it ends up destroying the very tissues and organs it was meant to protect. We tend to think of inflammation as synonymous with arthritis. However, inflammation can strike anywhere in your body. It's a major contributor to heart disease, cancer, Alzheimer's, and many more life-threatening conditions. Control inflammation and you control disease.

In her book, Prevent Cancer, Strokes, Heart Attacks and other Deadly Killers! Dr. Vijaya Nair, Harvard-trained M.D. FAMS, M.S. (Epid) says, "Inflammation is a double-edged sword. On the one hand, it protects you. When you get a splinter, inflammation makes sure that immune cells arrive at the scene of the injury to kill any bacteria or viruses that may have entered the broken skin. That's what all that redness, heat, swelling, and pain is about. Your immune system is going to war! On the other hand, inflammation can kill you. It can cause diseases no one wants to get, like cancer, arthritis, heart disease, digestive tract diseases, macular degeneration, Alzheimer's disease, and chronic fatigue syndrome."

Inflammation is only supposed to stick around as long as a threat exists. That's why your inflammation levels shoot through the roof if you have a severe bacterial infection, but, quickly go back to normal once the bacteria is successfully eliminated. When the body doesn't turn off the inflammation response -- when all those inflammatory chemicals stay in the system for a long time -- it ends up destroying the very tissues and organs it was meant to protect. Having high levels of inflammation for short periods of time is helpful, but having low levels of inflammation for long periods of time is deadly.

[truedelphi]

1. hawthorn Berry has long been seen by herbalists as a way to keep down

Parasites and inflammation regarding the heart.

Walnut tincture has been seen as a way to push inflammation leading to arthritis away from your body.

The herbalists have been talking about inflammation and all systems of the body for millennia.

[HysteryDiagnosis]

2. Very very good, here is a snippet from a German paper explaining

the potential role of inflammation in insulin resistance and obesity.

Low-Grade Inflammation, Obesity, and Insulin
Resistance in Adolescents
Christian Herder, Sophie Schneitler, Wolfgang Rathmann, Burkhard Haastert, Heiko Schneitler,
Horst Winkler, Renate Bredahl, Erik Hahnloser, and Stephan Martin
Institute for Clinical Diabetes Research (C.H., S.S., S.M.) and Institute of Biometrics and Epidemiology (W.R., B.H.),
German Diabetes Center, Leibniz Institute at Heinrich Heine University, 40225 Dusseldorf, Germany; and Gesundheitsamt
̈
(Public Health Office) (H.S., H.W., R.B., E.H.), 40227 Dusseldorf, Germany

IN ADULTS, NUMEROUS STUDIES have shown that a differential, subclinical, chronic activation of the immune system precedes the manifestation of type 2 diabetes mellitus (T2DM). Elevated systemic concentrations of specific acute phase proteins, cytokines, and chemokines as well as reduced
levels of the adipokine adiponectin predict the development of T2DM (1, 2). This association between low-grade inflammation and T2DM remains significant after adjustment for traditional risk factors so that it is reasonable to assume that low-grade inflammation is relevant in the pathogenesis of T2DM. However, some of the association between elevated levels of immune mediators and T2DM is also explained by obesity because adipose tissue has been shown to secrete many of the cytokines and chemokines that are considered T2DM risk factors (3–7).

The analysis of the role of low-grade inflammation in the development of insulin resistance and T2DM in children and adolescents is relevant because, in contrast to adults, it can be assumed that associations between low-grade inflammation, insulin resistance, and T2DM are not confounded by chronic
inflammatory conditions such as cardiovascular disease, arthritis, or bronchitis that are frequent comorbidities in T2DM patients or in old age. However, to the best of our knowledge, data
from prospective studies that investigate low-grade inflammation as a risk factor for T2DM in youth are not available. There is some evidence from case-control and cross-sectional surveys
that low-grade inflammation is associated with T2DM risk and insulin resistance in children and adolescents.

Most data come from studies that describe the positive association between C-
reactive protein (CRP) and obesity as a major risk factor for T2DM (8 –21). Only a few studies also investigated the relationship between CRP and glucose metabolism and found that
higher levels of CRP were associated with elevated fasting insulin levels and/or insulin resistance (14, 19, 20). These observations were extended in one study that showed that the
positive association between CRP and insulin is markedly attenuated after adjusting for BMI (14). This finding indicates that in youth as well as adults, the association between inflammation and T2DM risk cannot be explained by obesity alone and may support the development of T2DM at all ages.

Although CRP is widely considered a general marker of immune activation, cross-sectional studies show that CRP is only moderately or weakly correlated with many cytokines, chemokines, and adiponectin (22–24) and that the significant association between elevated levels of these mediators of innate
immunity and incident T2DM is independent of CRP levels (25,26). Thus, it can be expected that these markers represent different components of the immune system and that they provide
different information than CRP measurement. It is therefore interesting that several other reports suggested that also circulating concentrations of IL-6 (18, 21, 27), TNF␣ (16, 28), soluble
TNF␣ receptors (18, 28, 29), and E-selectin (21) are elevated in obesity and that IL-6 (27) and soluble TNF␣ receptor 2 may be associated with insulin resistance in children and adolescents
(29).

In addition, there are data from multiple adolescent populations on the inverse association between adiponectin and obesity and insulin resistance (30 –32). Adiponectin may be largely independent of systemic levels of many cytokines and chemokines (23) but has been shown to have strong anti-inflammatory effects on the molecular and cellular level (33, 34) so that it may also be considered as immune marker.

Taken together, there is growing evidence that obesity is associated with low-grade inflammation in youth, but data on the association with insulin resistance are limited to CRP and
soluble TNF␣ receptor 2. The objectives of this study were thus as follows: 1) to investigate the association between low-grade inflammation and measures of both obesity and glucose me-
tabolism by analyzing immune mediators that are expressed in adipocytes and may represent novel risk factors for the development of T2DM in adults, and 2) to examine whether the association between low-grade inflammation and insulin resistance is independent of obesity as assessed by body mass index (BMI) and waist circumference (WC).

To make a long story short, here is the conclusion:

Conclusions

In conclusion, high BMI and high WC are associated with a
specific pattern of low-grade immune activation in adolescents.
The same proinflammatory pattern is also seen in insulin-
resistant boys and girls. Obesity appears to mediate some of this
association. However, prospective studies will be necessary to
evaluate the relevance of subclinical inflammation for T2DM
risk in youth and allow comparisons of the role of this mech-
anism among children, adolescents, and adults.


̈

[truedelphi]

3. herbalists who understand inflammation and diabetes recommend

Cedar Berry, a remedy that costs pennies on the dollar compared to other remedies. Diabetics who have used Cedar Berry find their need for insulin drops about fifty percent.

Cedar Berry also helps with appetite suppression. Much easier to lose weight when not feeling hungry, and Cedar Berry fits that bill too.

[supernova]

4. What's the difference between Cedar berries and

juniper berries, i.e. the kind used to flavor liquors such as gin.

Just wondering.

[truedelphi]

5. Two separate types of trees -

Sort of like difference between apples and pears, or lemons and oranges.

Cedar berry is good for appetite control, and for diabetes.

Juniper berries are a tonic for ailing kidneys.

And like you say, they are used in making gin.

Anyone wanting to try out cedar berries - just google the terms "cedar berry" + purchase.

You can get like a pound (or maybe it is a half pound) of the ground up berries for under $ 20 - and that will last you months.

As with any herb, best to try a small amount to make sure there is not an allergy problem.

But usual doseage is one to two teaspoons per eight ounces of hot water. If you re diabetic, be sure and try it the first few times while at home and can monitor your sugar ups and downs - this can make you go low sugar so best to be at home.

Many users find themselves needing HALF AS MUCH insulin within the first few days of using this
tonic.

I use it for appetite control, and don't need to worry about my blood sugar levels.

[Name removed]

7. Deleted message

Message removed by moderator. Click here to review the message board rules.

[trotsky]

6. A two-year-old quack article?

Got all the hallmarks, though. Nice work.

* Oversimplification? Check. "Inflammation" is responsible for just about every single malady known to man. Always a hallmark of quackery - what good is your "cure" if it doesn't cure EVERYTHING?
* You are inadequate? Check. Telling you that your body isn't good enough - you gotta take herbs and supplements.
* Misleading credentials? Check. Dr. Nair's training is in Psychiatry!
* Quick and easy link to access the snake oil? Check! Oh look, Dr. Nair has her own site to peddle them! What a fortunate coincidence!

So the big question is, can I still use my Hulda Clark (who died of cancer) ZAPPER if I take these herbs? Will the magics strengthen each other or cancel each other out?

[truedelphi]

8. I wondered where you were, trotsky.

Hope you're having a nice December (no snark intended.) Season's greetings.

[trotsky]

9. Oh, I'm great!

Wondering why you haven't stopped in my recent thread? http://www.democraticunderground.com/discuss/duboard.php?az=view_all&address=222x96288

[truedelphi]

10. I'll K & R but will not comment

A family member was just diagnosed and I am too upset to think about this disease today.

Her outlook does not look good.

I am in total outraged numbness and fear. (Thirty six year old person, mom to two young kids.)